These findings offer a potential explanation for how rare heart inflammation cases might develop after vaccination.
However, researchers emphasize that the overall risk remains very low and that COVID-19 infection itself carries a higher risk of myocarditis and other complications.
The study also showed that blocking these inflammatory signals reduced heart-related damage in experimental settings, though it did not eliminate the broader immune response.
While compounds like genistein showed some protective effects in models, scientists stress that this is early research and not an immediate treatment recommendation.
